Vitamin E is your body's most abundant fat-soluble antioxidant, and the single most important antioxidant for heart attack prevention.
Research Demonstrates the Connection Between Vitamin E and Heart Disease
There is overwhelming evidence that vitamin E is an essential nutrient for heart attack prevention. As far back as 1933, Wilfred and Evan Shute, two cardiologists from Ontario, Canada, began using vitamin e to treat heart conditions. In their 1972 book, the Shutes reported having treated more than 30,000 patients with overwhelming positive results.
What makes vitamin E so valuable in preventing heart disease and atherosclerosis is that it can maneuver into fatty parts of the cell membrane that are inaccessible to the other network antioxidants.
The Cholesterol Myth
At the root of most heart problems is a condition called atherosclerosis, often referred to as "clogging" or "hardening" of the arteries. The commonly held belief is that your arteries get clogged because your cholesterol levels are too high. The medical community, and the public, seem to accept this as a scientific fact — except it is absolutely not true!
If you think that there is a connection between high cholesterol levels and heart disease, you need to watch this amazing presentation in which Dr. Malcolm Kendrick clearly demonstrates that there is absolutely NO correlation whatsoever.
High Cholesterol Levels Are Not What Causes Heart Disease
High cholesterol may accelerate the onset of atherosclerosis and heart disease, but it doesn't cause anything by itself. In fact, Vitamin E levels may even be a better predictor of heart attacks than are cholesterol levels. Low levels of vitamin E and hear disease almost always go hand-in-hand.
Heart Disease Starts with Oxidation. Cardiovascular disease begins when the walls of your arteries and the cholesterol in your blood are damaged by free radicals and oxidative stress. As a result, the arterial wall is no longer smooth, and it attracts more cholesterol that your liver is producing to try and repair the damage.
As the attacks from free radicals continue, the cholesterol oxidizes even more and begins to stick to the damaged arterial wall to form plaque. The arteries become narrower, diminishing the flow of blood to the heart. You now have atherosclerosis, the first step to heart disease.
It doesn't matter what your overall levels of cholesterol are — if the cholesterol gets damaged by free radical attacks, you will develop atherosclerosis.
To understand this more completely, watch another fascinating video in which Dr. Ron Rosedale exposes the cholesterol myth and reveals the truth about HDL and LDL-cholesterol and oxidation.
The most effective plan for heart attack prevention is a diet rich in antioxidants, especially vitamin E. Antioxidants prevent the free radical attacks to your arteries that cause atherosclerosis and almost all cardiovascular diseases.
The ultimate cause of heart disease is NOT your cholesterol level, it is the failure of the body's antioxidant network to do its job.
Go to the page on Heart Attack Prevention to learn about antioxidants that help in preventing heart disease.
Are You Taking Aspirin to Prevent Heart Attacks?
Vitamin E, as well as the herbs ginkgo biloba and garlic, have proven to be more effective and safer than aspirin, which is often recommended by doctors as a strategy for preventing heart disease.
The Hazards of Aspirin Consumption. The use of aspirin on a daily basis may cause stomach irritation that leads to bleeding, and possible kidney problems. Daily aspirin has also been connected to an increase in brain hemorrhages. Read the Wikipedia entry on the side effects of aspirin and other non-steroidal anti-inflammatory drugs (NAIDs).
Vitamin E, as well as ginkgo biloba and garlic offer safer alternatives, and come with other major benefits to boot. All of these natural supplements have been shown to prevent oxidation of LDL cholesterol, the fundamental cause of atherosclerosis. They also reduce your risk of stroke by making your blood platelets less sticky and therefore less likely to clot.
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